Nitric oxide (NO) is a soluble gas and signaling molecule synthesized continuously from L-arginine in various cell types. In blood vessels, NO maintains normal endothelial function through the modulation of vascular dilator tone, regulation of local cell growth and inflammatory processes, and the inhibition of blood platelet aggregation. Impaired synthesis and excessive degradation of NO are largely the result of increased oxidative stress, which contributes to endothelial dysfunction, hypertension, and an increased risk of life-threatening cardiovascular events. Supporting NO bioavailability in the blood vessels is an important consideration for blood pressure control and cardiovascular health.
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